| 1 | Sex differences in murine MASH induced by a fructose-palmitate-cholesterol-enriched diet | 4.5 | 11 | Citations (PDF) |
| 2 | Mechanistic underpinnings of AGEs-RAGE via DIAPH1 in ischemic, diabetic, and failing hearts | 3.5 | 7 | Citations (PDF) |
| 3 | Negative cooperativity regulates ligand activation of DIAPH1 and other diaphanous related formins | 4.4 | 5 | Citations (PDF) |
| 4 | Variations in weight loss and glycemic outcomes after sleeve gastrectomy by race and ethnicity | 4.0 | 1 | Citations (PDF) |
| 5 | The RAGE/DIAPH1 axis: mediator of obesity and proposed biomarker of human cardiometabolic disease | 5.5 | 14 | Citations (PDF) |
| 6 | RAGE/DIAPH1 and atherosclerosis through an evolving lens: Viewing the cell from the “Inside – Out” | 1.5 | 9 | Citations (PDF) |
| 7 | Continuous glucose monitoring captures glycemic variability in obesity after sleeve gastrectomy: A prospective cohort study | 1.9 | 4 | Citations (PDF) |
| 8 | Cardiometabolic disease: linking pathogenic mechanisms to therapeutic opportunities | 5.5 | 7 | Citations (PDF) |
| 9 | Introducing Socrates’ Corner to
ATVB
Journal: Critical Appraisals of Animal Models of Disease | 6.0 | 1 | Citations (PDF) |
| 10 | DIAPH1 mediates progression of atherosclerosis and regulates hepatic lipid metabolism in mice | 4.4 | 18 | Citations (PDF) |
| 11 | Pharmacological antagonism of receptor for advanced glycation end products signaling promotes thermogenesis, healthful body mass and composition, and metabolism in mice | 4.0 | 14 | Citations (PDF) |
| 12 | Involvement of the Receptor for Advanced Glycation End Products (RAGE) in high fat-high sugar diet-induced anhedonia in rats | 2.2 | 7 | Citations (PDF) |
| 13 | Disruption of the productive encounter complex results in dysregulation of DIAPH1 activity | 2.2 | 4 | Citations (PDF) |
| 14 | DIAPH1-MFN2 interaction regulates mitochondria-SR/ER contact and modulates ischemic/hypoxic stress | 13.7 | 49 | Citations (PDF) |
| 15 | Neuronal–glial communication perturbations in murine SOD1G93A spinal cord | 4.4 | 24 | Citations (PDF) |
| 16 | Soluble Receptor for Advanced Glycation End Products (sRAGE) Isoforms Predict Changes in Resting Energy Expenditure in Adults with Obesity during Weight Loss | 0.2 | 12 | Citations (PDF) |
| 17 | The RAGE/DIAPH1 Signaling Axis & Implications for the Pathogenesis of Diabetic Complications | 4.4 | 26 | Citations (PDF) |
| 18 | Glycation and a Spark of ALEs (Advanced Lipoxidation End Products) – Igniting RAGE/Diaphanous-1 and Cardiometabolic Disease | 2.4 | 18 | Citations (PDF) |
| 19 | Journey to a Receptor for Advanced Glycation End Products Connection in Severe Acute Respiratory Syndrome Coronavirus 2 Infection | 6.0 | 26 | Citations (PDF) |
| 20 | Inflammation Meets Metabolism Roles: for the Receptor for Advanced Glycation End Products Axis in Cardiovascular Disease | 2.8 | 20 | Citations (PDF) |
| 21 | Aldose Reductase: An Emerging Target for Development of Interventions for Diabetic Cardiovascular Complications | 3.8 | 93 | Citations (PDF) |
| 22 | MicroRNA-33 Inhibits Adaptive Thermogenesis and Adipose Tissue Beiging | 6.0 | 21 | Citations (PDF) |
| 23 | Microglia RAGE exacerbates the progression of neurodegeneration within the SOD1G93A murine model of amyotrophic lateral sclerosis in a sex-dependent manner | 9.0 | 32 | Citations (PDF) |
| 24 | Diabetes and Cardiovascular Complications: The Epidemics Continue | 2.9 | 19 | Citations (PDF) |
| 25 | AGE/RAGE/DIAPH1 axis is associated with immunometabolic markers and risk of insulin resistance in subcutaneous but not omental adipose tissue in human obesity | 3.0 | 25 | Citations (PDF) |
| 26 | Microbial signatures in the lower airways of mechanically ventilated COVID-19 patients associated with poor clinical outcome | 16.0 | 174 | Citations (PDF) |
| 27 | Silencing Myeloid Netrin-1 Induces Inflammation Resolution and Plaque Regression | 13.1 | 43 | Citations (PDF) |
| 28 | A pilot open-label study of aldose reductase inhibition with AT-001 (caficrestat) in patients hospitalized for COVID-19 infection: Results from a registry-based matched-control analysis | 3.0 | 13 | Citations (PDF) |
| 29 | Small-molecule antagonism of the interaction of the RAGE cytoplasmic domain with DIAPH1 reduces diabetic complications in mice | 12.5 | 74 | Citations (PDF) |
| 30 | Advanced Glycation End Products: Building on the Concept of the “Common Soil” in Metabolic Disease | 2.5 | 164 | Citations (PDF) |
| 31 | Annual Report on Sex in Preclinical Studies | 6.0 | 10 | Citations (PDF) |
| 32 | RAGE Mediates Cholesterol Efflux Impairment in Macrophages Caused by Human Advanced Glycated Albumin | 4.4 | 15 | Citations (PDF) |
| 33 | Chronic low-dose rapamycin treatment fine tunes cardioprotective signalling in ischaemia-reperfused diabetic hearts | 5.5 | 0 | Citations (PDF) |
| 34 | Receptor for Advanced Glycation End Products is Involved in Platelet Hyperactivation and Arterial Thrombosis during Chronic Kidney Disease | 4.1 | 11 | Citations (PDF) |
| 35 | Multiomics of World Trade Center Particulate Matter–induced Persistent Airway Hyperreactivity. Role of Receptor for Advanced Glycation End Products | 3.8 | 15 | Citations (PDF) |
| 36 | An Eclectic Cast of Cellular Actors Orchestrates Innate Immune Responses in the Mechanisms Driving Obesity and Metabolic Perturbation | 13.1 | 24 | Citations (PDF) |
| 37 | Leukocyte Heterogeneity in Adipose Tissue, Including in Obesity | 13.1 | 66 | Citations (PDF) |
| 38 | S100A9-RAGE Axis Accelerates Formation of Macrophage-Mediated Extracellular Vesicle Microcalcification in Diabetes Mellitus | 6.0 | 92 | Citations (PDF) |
| 39 | Receptor for Advanced Glycation End Products (RAGE) and Mechanisms and Therapeutic Opportunities in Diabetes and Cardiovascular Disease: Insights From Human Subjects and Animal Models | 2.4 | 192 | Citations (PDF) |
| 40 | RAGE impairs murine diabetic atherosclerosis regression and implicates IRF7 in macrophage inflammation and cholesterol metabolism | 5.4 | 60 | Citations (PDF) |
| 41 | Incense Burning is Associated with Human Oral Microbiota Composition | 3.4 | 16 | Citations (PDF) |
| 42 | A Receptor of the Immunoglobulin Superfamily Regulates Adaptive Thermogenesis | 6.3 | 50 | Citations (PDF) |
| 43 | Metabolism, Obesity, and Diabetes Mellitus | 6.0 | 18 | Citations (PDF) |
| 44 | Metabolic dysfunction in Emirati subjects in Abu Dhabi: Relationship to levels of soluble RAGEs | 1.0 | 4 | Citations (PDF) |
| 45 | The Receptor for Advanced Glycation End Products (RAGE) and DIAPH1: Implications for vascular and neuroinflammatory dysfunction in disorders of the central nervous system | 3.5 | 64 | Citations (PDF) |
| 46 | The rationale and design of the personal diet study, a randomized clinical trial evaluating a personalized approach to weight loss in individuals with pre-diabetes and early-stage type 2 diabetes | 1.6 | 25 | Citations (PDF) |
| 47 | Diabetes Mellitus and Cardiovascular Disease | 6.0 | 159 | Citations (PDF) |
| 48 | Netrin-1 Alters Adipose Tissue Macrophage Fate and Function in Obesity | 2.8 | 48 | Citations (PDF) |
| 49 | Imaging VEGF Receptors and αvβ3 Integrins in a Mouse Hindlimb Ischemia Model of Peripheral Arterial Disease | 2.2 | 3 | Citations (PDF) |
| 50 | The receptor for advanced glycation endproducts is a mediator of toxicity by IAPP and other proteotoxic aggregates: Establishing and exploiting common ground for novel amyloidosis therapies | 5.9 | 25 | Citations (PDF) |
| 51 | Highlighting Diabetes Mellitus | 6.0 | 260 | Citations (PDF) |
| 52 | Analysis of the Role of the Conserved Disulfide in Amyloid Formation by Human Islet Amyloid Polypeptide in Homogeneous and Heterogeneous Environments | 2.4 | 24 | Citations (PDF) |
| 53 | Myeloid ATG16L1 does not affect adipose tissue inflammation or body mass in mice fed high fat diet | 1.6 | 7 | Citations (PDF) |
| 54 | The Receptor for Advanced Glycation Endproducts (RAGE) and Mediation of Inflammatory Neurodegeneration 2018, 08, | | 71 | Citations (PDF) |
| 55 | Reporting Sex and Sex Differences in Preclinical Studies | 6.0 | 18 | Citations (PDF) |
| 56 | Response by Daugherty et al to Letter Regarding Article, “Consideration of Sex Differences in Design and Reporting of Experimental Arterial Pathology Studies: A Statement From the
<i>Arteriosclerosis, Thrombosis, and Vascular Biology</i>
Council” | 6.0 | 3 | Citations (PDF) |
| 57 | Types of tobacco consumption and the oral microbiome in the United Arab Emirates Healthy Future (UAEHFS) Pilot Study | 3.4 | 57 | Citations (PDF) |
| 58 | Diaphanous 1 (DIAPH1) is Highly Expressed in the Aged Human Medial Temporal Cortex and Upregulated in Myeloid Cells During Alzheimer’s Disease | 2.6 | 20 | Citations (PDF) |
| 59 | The UAE healthy future study: a pilot for a prospective cohort study of 20,000 United Arab Emirates nationals | 3.1 | 46 | Citations (PDF) |
| 60 | Amyloidogenicity, Cytotoxicity, and Receptor Activity of Bovine Amylin: Implications for Xenobiotic Transplantation and the Design of Nontoxic Amylin Variants | 3.7 | 24 | Citations (PDF) |
| 61 | Deletion of the forminDiaph1protects from structural and functional abnormalities in the murine diabetic kidney | 3.3 | 26 | Citations (PDF) |
| 62 | Soluble levels of receptor for advanced glycation endproducts and dysfunctional high-density lipoprotein in persons infected with human immunodeficiency virus | 1.2 | 3 | Citations (PDF) |
| 63 | Patterns of tobacco use in the United Arab Emirates Healthy Future (UAEHFS) pilot study | 2.3 | 43 | Citations (PDF) |
| 64 | RAGE binds preamyloid IAPP intermediates and mediates pancreatic β cell proteotoxicity | 10.6 | 80 | Citations (PDF) |
| 65 | Small Molecule Antagonists of RAGE‐DIAPH1: Novel Therapeutic Opportunities in Metabolic and Chronic Disease | 0.6 | 0 | Citations (PDF) |
| 66 | RAGE-Mediated Suppression of Interleukin-10 Results in Enhanced Mortality in a Murine Model of Acinetobacter baumannii Sepsis | 2.7 | 31 | Citations (PDF) |
| 67 | 2016
ATVB
Plenary Lecture | 6.0 | 47 | Citations (PDF) |
| 68 | Evolutionary Adaptation and Amyloid Formation: Does the Reduced Amyloidogenicity and Cytotoxicity of Ursine Amylin Contribute to the Metabolic Adaption of Bears and Polar Bears? | 2.0 | 15 | Citations (PDF) |
| 69 | Ager
Deletion Enhances Ischemic Muscle Inflammation, Angiogenesis, and Blood Flow Recovery in Diabetic Mice | 6.0 | 39 | Citations (PDF) |
| 70 | Glycation & the RAGE axis: targeting signal transduction through DIAPH1 | 2.0 | 31 | Citations (PDF) |
| 71 | The AGE-RAGE axis in an Arab population: The United Arab Emirates Healthy Futures (UAEHFS) pilot study | 1.0 | 7 | Citations (PDF) |
| 72 | Diabetes Exacerbates Infection via Hyperinflammation by Signaling through TLR4 and RAGE | 4.4 | 68 | Citations (PDF) |
| 73 | Advanced glycation end products receptor RAGE controls myocardial dysfunction and oxidative stress in high-fat fed mice by sustaining mitochondrial dynamics and autophagy-lysosome pathway | 3.7 | 66 | Citations (PDF) |
| 74 | The Formin, DIAPH1, is a Key Modulator of Myocardial Ischemia/Reperfusion Injury | 9.7 | 40 | Citations (PDF) |
| 75 | [O1–14–04]: RAGE AND DIAPH‐1 REGULATE CRITICAL PHENOTYPES OF MICROGLIA IN HEALTHY AGING AND ALZHEIMER'S DISEASE | 0.5 | 0 | Citations (PDF) |
| 76 | The AGE-RAGE Axis: Implications for Age-Associated Arterial Diseases | 2.3 | 147 | Citations (PDF) |
| 77 | Aldose reductase modulates acute activation of mesenchymal markers via the β-catenin pathway during cardiac ischemia-reperfusion | 2.3 | 4 | Citations (PDF) |
| 78 | Receptor for advanced glycation end-products and World Trade Center particulate induced lung function loss: A case-cohort study and murine model of acute particulate exposure | 2.3 | 35 | Citations (PDF) |
| 79 | Translational Approach to Understanding RAGE pathway in Acute respiratory distress syndrome | 0.1 | 0 | Citations (PDF) |
| 80 | Islet Amyloid Polypeptide: Structure, Function, and Pathophysiology | 5.5 | 242 | Citations (PDF) |
| 81 | Soluble RAGE Treatment Delays Progression of Amyotrophic Lateral Sclerosis in SOD1 Mice | 3.4 | 38 | Citations (PDF) |
| 82 | Soluble Receptor for Advanced Glycation End Products Improves Stromal Cell–Derived Factor-1 Activity in Model Diabetic Environments | 5.3 | 17 | Citations (PDF) |
| 83 | Small Molecule Inhibition of Ligand-Stimulated RAGE-DIAPH1 Signal Transduction | 3.4 | 107 | Citations (PDF) |
| 84 | Soluble Levels of Receptor for Advanced Glycation Endproducts (RAGE) and Progression of Atherosclerosis in Individuals Infected with Human Immunodeficiency Virus: ACTG NWCS 332 | 4.0 | 6 | Citations (PDF) |
| 85 | Aldose Reductase Acts as a Selective Derepressor of PPARγ and the Retinoic Acid Receptor | 6.3 | 29 | Citations (PDF) |
| 86 | Three-year follow-up comparing metabolic surgery versus medical weight management in patients with type 2 diabetes and BMI 30–35. The role of sRAGE biomarker as predictor of satisfactory outcomes | 2.5 | 29 | Citations (PDF) |
| 87 | Cellular mechanisms and consequences of glycation in atherosclerosis and obesity | 4.1 | 63 | Citations (PDF) |
| 88 | Change in the Molecular Dimension of a RAGE-Ligand Complex Triggers RAGE Signaling | 3.8 | 60 | Citations (PDF) |
| 89 | Mechanisms of transcription factor acetylation and consequences in hearts | 4.1 | 29 | Citations (PDF) |
| 90 | Complying With the National Institutes of Health Guidelines and Principles for Rigor and Reproducibility | 6.0 | 13 | Citations (PDF) |
| 91 | The multiple faces of RAGE – opportunities for therapeutic intervention in aging and chronic disease | 3.7 | 96 | Citations (PDF) |
| 92 | Emerging Targets for Therapeutic Development in Diabetes and Its Complications: The RAGE Signaling Pathway | 4.7 | 98 | Citations (PDF) |
| 93 | Population-Level Prediction of Type 2 Diabetes From Claims Data and Analysis of Risk Factors | 1.6 | 199 | Citations (PDF) |
| 94 | Receptor for Advanced Glycation End Products and its Inflammatory Ligands are Upregulated in Amyotrophic Lateral Sclerosis | 3.4 | 64 | Citations (PDF) |
| 95 | The Growing Problem of Obesity | 6.0 | 16 | Citations (PDF) |
| 96 | Soluble RAGEs — Prospects for treating & tracking metabolic and inflammatory disease | 2.5 | 95 | Citations (PDF) |
| 97 | Treatment effect with anti-RAGE F(ab′)2 antibody improves hind limb angiogenesis and blood flow in Type 1 diabetic mice with left femoral artery ligation | 2.4 | 16 | Citations (PDF) |
| 98 | RAGE Suppresses ABCG1-Mediated Macrophage Cholesterol Efflux in Diabetes | 4.2 | 65 | Citations (PDF) |
| 99 | Deletion of mDia1 is Protective Against Renal Damage in a Murine Model of Diabetes | 0.6 | 0 | Citations (PDF) |
| 100 | Randomized Pilot Trial of Bariatric Surgery Versus Intensive Medical Weight Management on Diabetes Remission in Type 2 Diabetic Patients Who Do NOT Meet NIH Criteria for Surgery and the Role of Soluble RAGE as a Novel Biomarker of Success | 4.6 | 111 | Citations (PDF) |
| 101 | Unlocking the biology of RAGE in diabetic microvascular complications | 8.5 | 192 | Citations (PDF) |
| 102 | Receptor for advanced glycation end products and its ligand high-mobility group box-1 mediate allergic airway sensitization and airway inflammation | 6.1 | 149 | Citations (PDF) |
| 103 | RAGE Regulates the Metabolic and Inflammatory Response to High-Fat Feeding in Mice | 4.2 | 198 | Citations (PDF) |
| 104 | Imaging RAGE expression in atherosclerotic plaques in hyperlipidemic pigs | 2.6 | 12 | Citations (PDF) |
| 105 | Recent Highlights of
ATVB | 6.0 | 11 | Citations (PDF) |
| 106 | Reduced expression of Munc13-1 in human and porcine diabetic peripheral nerve | 2.3 | 2 | Citations (PDF) |
| 107 | The Receptor for Advanced Glycation End Products (RAGE) Affects T Cell Differentiation in OVA Induced Asthma | 2.3 | 45 | Citations (PDF) |
| 108 | Receptor for AGE (RAGE) Expressed in Macrophages and Neurons Regulates Peripheral Nerve Repair after Injury (P1.279) | 1.0 | 0 | Citations (PDF) |
| 109 | Imaging receptor for advanced glycation end product expression in mouse model of hind limb ischemia | 2.6 | 17 | Citations (PDF) |
| 110 | Macrophages | 6.0 | 1 | Citations (PDF) |
| 111 | Islet amyloid polypeptide toxicity and membrane interactions | 7.5 | 142 | Citations (PDF) |
| 112 | Radical Roles for RAGE in the Pathogenesis of Oxidative Stress in Cardiovascular Diseases and Beyond | 4.4 | 211 | Citations (PDF) |
| 113 | The receptor for advanced glycation end products mediates lung endothelial activation by RBCs | 3.2 | 25 | Citations (PDF) |
| 114 | Mechanisms of islet amyloidosis toxicity in type 2 diabetes | 2.7 | 188 | Citations (PDF) |
| 115 | Carbon monoxide form of PEGylated hemoglobin protects myocardium against ischemia/reperfusion injury in diabetic and normal mice | 3.8 | 38 | Citations (PDF) |
| 116 | Combinatorial Library of Improved Peptide Aptamers, CLIPs to Inhibit RAGE Signal Transduction in Mammalian Cells | 2.3 | 25 | Citations (PDF) |
| 117 | Microglial RAGE Accelerates Mortality and Neuronal Dysfunction in a Murine Model of Familial Amyotrophic Lateral Sclerosis (P02.173) | 1.0 | 0 | Citations (PDF) |
| 118 | Rational design of potent domain antibody inhibitors of amyloid fibril assembly | 7.5 | 104 | Citations (PDF) |
| 119 | Formin mDia1 Mediates Vascular Remodeling via Integration of Oxidative and Signal Transduction Pathways | 13.1 | 90 | Citations (PDF) |
| 120 | Insulin Resistance and Metabolic Syndrome | 6.0 | 8 | Citations (PDF) |
| 121 | The diverse ligand repertoire of the receptor for advanced glycation endproducts and pathways to the complications of diabetes | 2.5 | 145 | Citations (PDF) |
| 122 | Sensitivity of Amyloid Formation by Human Islet Amyloid Polypeptide to Mutations at Residue 20 | 4.1 | 84 | Citations (PDF) |
| 123 | Glycation and Insulin Resistance | 6.0 | 70 | Citations (PDF) |
| 124 | Imaging of Receptors for Advanced Glycation End Products in Experimental Myocardial Ischemia and Reperfusion Injury | 6.1 | 16 | Citations (PDF) |
| 125 | Signal Transduction in Receptor for Advanced Glycation End Products (RAGE) | 2.2 | 117 | Citations (PDF) |
| 126 | Receptor for Advanced Glycation End Products (RAGE) and Implications for the Pathophysiology of Heart Failure | 2.9 | 77 | Citations (PDF) |
| 127 | RAGE binds C1q and enhances C1q-mediated phagocytosis | 2.5 | 68 | Citations (PDF) |
| 128 | RAGE Expression in Human T Cells: A Link between Environmental Factors and Adaptive Immune Responses | 2.3 | 96 | Citations (PDF) |
| 129 | Alternative splicing of RAGE: roles in biology and disease | 5.8 | 84 | Citations (PDF) |
| 130 | Advanced Glycation End Product Recognition by the Receptor for AGEs | 3.8 | 203 | Citations (PDF) |
| 131 | Imaging the effect of receptor for advanced glycation endproducts on angiogenic response to hindlimb ischemia in diabetes | 2.6 | 8 | Citations (PDF) |
| 132 | Aldose reductase pathway contributes to vulnerability of aging myocardium to ischemic injury | 3.7 | 20 | Citations (PDF) |
| 133 | Human Vascular Endothelial Cells: A Model System for Studying Vascular Inflammation in Diabetes and Atherosclerosis | 5.1 | 174 | Citations (PDF) |
| 134 | Improvement in Angiogenesis and Restoration of Blood Flow in Diabetic Mice by Sanguinate
TM | 0.6 | 1 | Citations (PDF) |
| 135 | Polyol pathway and RAGE: a central metabolic and signaling axis in diabetic complications | 2.9 | 5 | Citations (PDF) |
| 136 | RAGE Modulates Hypoxia/Reoxygenation Injury in Adult Murine Cardiomyocytes via JNK and GSK-3β Signaling Pathways | 2.3 | 86 | Citations (PDF) |
| 137 | The RAGE Axis | 13.1 | 369 | Citations (PDF) |
| 138 | Deletion of the Receptor for Advanced Glycation End Products Reduces Glomerulosclerosis and Preserves Renal Function in the Diabetic OVE26 Mouse | 4.2 | 163 | Citations (PDF) |
| 139 | Advanced Glycation End Product (AGE)-Receptor for AGE (RAGE) Signaling and Up-regulation of Egr-1 in Hypoxic Macrophages | 2.2 | 113 | Citations (PDF) |
| 140 | RAGE and the pathogenesis of chronic kidney disease | 32.8 | 128 | Citations (PDF) |
| 141 | Advanced glycation end-products: Implications for diabetic and non-diabetic nephropathies | 3.5 | 120 | Citations (PDF) |
| 142 | RAGE, glomerulosclerosis and proteinuria: Roles in podocytes and endothelial cells | 8.5 | 50 | Citations (PDF) |
| 143 | Receptor for advanced glycation endproducts mediates pro-atherogenic responses to periodontal infection in vascular endothelial cells | 1.5 | 41 | Citations (PDF) |
| 144 | Ultrastructural Features of Retinal Capillary Basement Membrane Thickening in Diabetic Swine | 1.3 | 45 | Citations (PDF) |
| 145 | Advanced glycation endproducts: from precursors to RAGE: round and round we go | 2.3 | 150 | Citations (PDF) |
| 146 | Deciphering the complications of diabetes and obesity: the keystone to identification of therapeutic targets | 2.9 | 0 | Citations (PDF) |
| 147 | Intra-coronary administration of soluble receptor for advanced glycation end-products attenuates cardiac remodeling with decreased myocardial transforming growth factor-β1 expression and fibrosis in minipigs with ischemia-reperfusion injury | 4.4 | 19 | Citations (PDF) |
| 148 | Tempering the wrath of RAGE: An emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation | 3.8 | 125 | Citations (PDF) |
| 149 | Mice deficient in PKC β and apolipoprotein E display decreased atherosclerosis | 0.6 | 74 | Citations (PDF) |
| 150 | Alternative splicing of the murine receptor for advanced glycation end‐products (RAGE) gene | 0.6 | 101 | Citations (PDF) |
| 151 | Controllable Expansion of Primary Cardiomyocytes by Reversible Immortalization | 3.2 | 25 | Citations (PDF) |
| 152 | Receptor for AGE (RAGE) and its ligands—cast into leading roles in diabetes and the inflammatory response | 3.7 | 218 | Citations (PDF) |
| 153 | Therapies for hyperglycaemia-induced diabetic complications: from animal models to clinical trials | 79.7 | 318 | Citations (PDF) |
| 154 | Porphyromonas gingivalis infection and prothrombotic effects in human aortic smooth muscle cells | 2.3 | 30 | Citations (PDF) |
| 155 | Neuronal RAGE expression modulates severity of injury following transient focal cerebral ischemia | 1.6 | 47 | Citations (PDF) |
| 156 | Receptor for advanced glycation end product (RAGE)-dependent modulation of early growth response-1 in hepatic ischemia/reperfusion injury | 4.2 | 74 | Citations (PDF) |
| 157 | RAGE: therapeutic target and biomarker of the inflammatory response—the evidence mounts | 2.9 | 252 | Citations (PDF) |
| 158 | Anti-Receptor for Advanced Glycation End Products Therapies as Novel Treatment for Abdominal Aortic Aneurysm | 4.6 | 62 | Citations (PDF) |
| 159 | The expression of the receptor for glycation endproducts (RAGE) in oral squamous cell carcinomas | 1.2 | 18 | Citations (PDF) |
| 160 | Mechanisms of Disease: advanced glycation end-products and their receptor in inflammation and diabetes complications | 6.0 | 391 | Citations (PDF) |
| 161 | Oxygen Deprivation Triggers Upregulation of Early Growth Response-1 by the Receptor for Advanced Glycation End Products | 13.1 | 105 | Citations (PDF) |
| 162 | Interaction of the RAGE Cytoplasmic Domain with Diaphanous-1 Is Required for Ligand-stimulated Cellular Migration through Activation of Rac1 and Cdc42 | 2.2 | 331 | Citations (PDF) |
| 163 | Soluble receptor for advanced glycation end products (sRAGE) and endogenous secretory RAGE (esRAGE) in amniotic fluid: modulation by infection and inflammation | 1.2 | 55 | Citations (PDF) |
| 164 | AGE/RAGE produces endothelial dysfunction in coronary arterioles in Type 2 diabetic mice | 3.5 | 162 | Citations (PDF) |
| 165 | RAGE modulates myocardial injury consequent to LAD infarction via impact on JNK and STAT signaling in a murine model | 3.5 | 130 | Citations (PDF) |
| 166 | Identification, classification, and expression of
RAGE
gene splice variants | 0.6 | 346 | Citations (PDF) |
| 167 | Development of Receptor for Advanced Glycation End Products–Directed Imaging of Atherosclerotic Plaque in a Murine Model of Spontaneous Atherosclerosis | 2.9 | 26 | Citations (PDF) |
| 168 | Vascular and inflammatory stresses mediate atherosclerosis via RAGE and its ligands in apoE–/– mice | 10.6 | 344 | Citations (PDF) |
| 169 | Hypoxia-inducible Factor-1 Mediates Neuronal Expression of the Receptor for Advanced Glycation End Products following Hypoxia/Ischemia | 2.2 | 94 | Citations (PDF) |
| 170 | RAGE Ligand Upregulation of VEGF Secretion in ARPE-19 Cells 2007, 48, 1355 | | 110 | Citations (PDF) |
| 171 | The RAGE connection to diabetes and atherosclerosis: an intertwined web of advanced glycation and inflammation | 0.8 | 4 | Citations (PDF) |
| 172 | Infection with a periodontal pathogen increases mononuclear cell adhesion to human aortic endothelial cells | 1.5 | 102 | Citations (PDF) |
| 173 | RAGE: Exacting a toll on the host in response to polymicrobial sepsis and Listeria monocytogenes | 6.0 | 8 | Citations (PDF) |
| 174 | Vascular endothelial sampling and analysis of gene transcripts: a new quantitative approach to monitor vascular inflammation | 2.8 | 27 | Citations (PDF) |
| 175 | Porphyromonas gingivalisinfection and cell death in human aortic endothelial cells | 1.9 | 55 | Citations (PDF) |
| 176 | RAGE Activation by S100P in Colon Cancer Stimulates Growth, Migration, and Cell Signaling Pathways | 1.7 | 141 | Citations (PDF) |
| 177 | The biology of RAGE and its ligands: Uncovering mechanisms at the heart of diabetes and its complications | 5.1 | 53 | Citations (PDF) |
| 178 | RAGE modulates vascular inflammation and atherosclerosis in a murine model of type 2 diabetes | 1.5 | 227 | Citations (PDF) |
| 179 | Receptor for Advanced Glycation End Products and the Cardiovascular Complications of Diabetes and Beyond: Lessons from AGEing | 3.5 | 21 | Citations (PDF) |
| 180 | Glycation and RAGE: Common Links in the Pathogenesis of Microvascular and Macrovascular Complications of Diabetes | 1.8 | 4 | Citations (PDF) |
| 181 | Protein Kinase C β/Early Growth Response-1 Pathway | 2.3 | 65 | Citations (PDF) |
| 182 | Advanced Glycation End Products | 18.0 | 2,097 | Citations (PDF) |
| 183 | The ligand/RAGE axis: Lighting the fuse and igniting vascular stress | 4.7 | 18 | Citations (PDF) |
| 184 | Induction of inflammatory bowel disease accelerates adenoma formation in Min +/− mice | 1.8 | 20 | Citations (PDF) |
| 185 | The role of RAGE in the pathogenesis of intestinal barrier dysfunction after hemorrhagic shock | 3.2 | 65 | Citations (PDF) |
| 186 | Receptor for Advanced-Glycation End Products | 18.0 | 216 | Citations (PDF) |
| 187 | The RAGE Axis and Endothelial Dysfunction: Maladaptive Roles in the Diabetic Vasculature and Beyond | 7.2 | 82 | Citations (PDF) |
| 188 | Influence of ischemic injury on vein graft remodeling: Role of cyclic adenosine monophosphate second messenger pathway in enhanced vein graft preservation | 2.6 | 18 | Citations (PDF) |
| 189 | At Least 2 Distinct Pathways Generating Reactive Oxygen Species Mediate Vascular Cell Adhesion Molecule-1 Induction by Advanced Glycation End Products | 6.0 | 205 | Citations (PDF) |
| 190 | Central Role of PKCβ in Neointimal Expansion Triggered by Acute Arterial Injury | 13.1 | 26 | Citations (PDF) |
| 191 | RAGE limits regeneration after massive liver injury by coordinated suppression of TNF-α and NF-κB | 9.3 | 136 | Citations (PDF) |
| 192 | Diabetic Vascular Disease: It's All the RAGE | 6.3 | 45 | Citations (PDF) |
| 193 | Mechanisms for the induction of HNE- MDA- and AGE-adducts, RAGE and VEGF in retinal pigment epithelial cells | 2.5 | 118 | Citations (PDF) |
| 194 | The RAGE Gly82Ser polymorphism is not associated with cardiovascular disease in the Framingham offspring study | 1.5 | 45 | Citations (PDF) |
| 195 | Advanced glycation end products and RAGE: a common thread in aging, diabetes, neurodegeneration, and inflammation | 2.2 | 763 | Citations (PDF) |
| 196 | Title is missing! | 3.9 | 27 | Citations (PDF) |
| 197 | RAGE: A journey from the complications of diabetes to disorders of the nervous system – striking a fine balance between injury and repair | 0.7 | 6 | Citations (PDF) |
| 198 | S100P Stimulates Cell Proliferation and Survival via Receptor for Activated Glycation End Products (RAGE) | 2.2 | 241 | Citations (PDF) |
| 199 | Protein Glycation | 13.1 | 409 | Citations (PDF) |
| 200 | Early Growth Response-1 Promotes Atherogenesis | 13.1 | 148 | Citations (PDF) |
| 201 | Antagonism of RAGE suppresses peripheral nerve regeneration | 0.6 | 105 | Citations (PDF) |
| 202 | RAGE Axis | 6.0 | 134 | Citations (PDF) |
| 203 | RAGE: A Novel Target for Drug Intervention in Diabetic Vascular Disease | 3.7 | 74 | Citations (PDF) |
| 204 | Porphyromonas gingivalisinduces its uptake by human macrophages and promotes foam cell formation in vitro | 1.9 | 81 | Citations (PDF) |
| 205 | Advanced glycation end products and vascular inflammation: implications for accelerated atherosclerosis in diabetes | 5.5 | 881 | Citations (PDF) |
| 206 | PKCβ regulates ischemia/reperfusion injury in the lung | 10.6 | 52 | Citations (PDF) |
| 207 | Receptor for advanced glycation end products (RAGE) regulates sepsis but not the adaptive immune response | 10.6 | 446 | Citations (PDF) |
| 208 | PKCβ regulates ischemia/reperfusion injury in the lung | 10.6 | 6 | Citations (PDF) |
| 209 | RAGE mediates amyloid-β peptide transport across the blood-brain barrier and accumulation in brain | 33.0 | 1,410 | Citations (PDF) |
| 210 | RAGE Drives the Development of Glomerulosclerosis and Implicates Podocyte Activation in the Pathogenesis of Diabetic Nephropathy | 3.4 | 574 | Citations (PDF) |
| 211 | Central role of RAGE-dependent neointimal expansion in arterial restenosis | 10.6 | 291 | Citations (PDF) |
| 212 | Glycation, Inflammation, and RAGE | 13.1 | 501 | Citations (PDF) |
| 213 | Oral Infection With a Periodontal Pathogen Accelerates Early Atherosclerosis in Apolipoprotein E–Null Mice | 6.0 | 352 | Citations (PDF) |
| 214 | Synergistic action of advanced glycation end products and endogenous nitric oxide leads to neuronal apoptosis in vitro: A new insight into selective nitrergic neuropathy in diabetes | 7.5 | 103 | Citations (PDF) |
| 215 | Central role of RAGE-dependent neointimal expansion in arterial restenosis | 10.6 | 196 | Citations (PDF) |
| 216 | Receptor for advanced glycation endproducts (RAGE) and the complications of diabetes | 11.6 | 263 | Citations (PDF) |
| 217 | Receptor for advanced glycation endproducts: a multiligand receptor magnifying cell stress in diverse pathologic settings | 15.4 | 261 | Citations (PDF) |
| 218 | Receptor for advanced glycation endproducts (RAGE) and vascular inflammation: Insights into the pathogenesis of macrovascular complications in diabetes | 4.7 | 172 | Citations (PDF) |
| 219 | RAGE is a multiligand receptor of the immunoglobulin superfamily: implications for homeostasis and chronic disease | 5.5 | 286 | Citations (PDF) |
| 220 | RAGE and arthritis: the G82S polymorphism amplifies the inflammatory response | 3.8 | 356 | Citations (PDF) |
| 221 | Receptor for Advanced Glycation End Products on Human Synovial Fibroblasts | 0.4 | 44 | Citations (PDF) |
| 222 | Receptor for Advanced Glycation End Products, Inflammation, and Accelerated Periodontal Disease in Diabetes: Mechanisms and Insights Into Therapeutic Modalities 2001, 6, 113-118 | | 147 | Citations (PDF) |
| 223 | Involvement of Microglial Receptor for Advanced Glycation Endproducts (RAGE) in Alzheimer's Disease: Identification of a Cellular Activation Mechanism | 4.0 | 413 | Citations (PDF) |
| 224 | Blockade of Receptor for Advanced Glycation End-Products Restores Effective Wound Healing in Diabetic Mice | 3.4 | 414 | Citations (PDF) |
| 225 | Key signaling pathways regulate the biological activities and accumulation of amyloid-β | 3.4 | 15 | Citations (PDF) |
| 226 | Receptor for age (RAGE) is a gene within the major histocompatibility class III region: implications for host response mechanisms in homeostasis and chronic disease | 5.8 | 66 | Citations (PDF) |
| 227 | Activation of NADPH oxidase by AGE links oxidant stress to altered gene expression via RAGE | 3.0 | 968 | Citations (PDF) |
| 228 | β2-Microglobulin modified with advanced glycation end products delays monocyte apoptosis | 5.3 | 72 | Citations (PDF) |
| 229 | Colonoscopy in Mice | 2.3 | 40 | Citations (PDF) |
| 230 | Hyperglycemia, glycoxidation and receptor for advanced glycation endproducts: potential mechanisms underlying diabetic complications, including diabetes-associated periodontitis | 14.8 | 139 | Citations (PDF) |
| 231 | Blockade of RAGE–amphoterin signalling suppresses tumour growth and metastases | 37.9 | 1,174 | Citations (PDF) |
| 232 | Atherosclerosis and diabetes: The rage connection | 4.7 | 177 | Citations (PDF) |
| 233 | β-Amyloid-induced migration of monocytes across human brain endothelial cells involves RAGE and PECAM-1 | 4.2 | 211 | Citations (PDF) |
| 234 | Blockade of RAGE suppresses periodontitis-associated bone loss in diabetic mice | 10.6 | 327 | Citations (PDF) |
| 235 | RAGE: A Multiligand Receptor Contributing to the Cellular Response in Diabetic Vasculopathy and Inflammation | 2.5 | 125 | Citations (PDF) |
| 236 | The biology of the receptor for advanced glycation end products and its ligands | 3.6 | 617 | Citations (PDF) |
| 237 | Cellular cofactors potentiating induction of stress and cytotoxicity by amyloid β-peptide | 4.1 | 56 | Citations (PDF) |
| 238 | RAGE: A New Target for the Prevention and Treatment of the Vascular and Inflammatory Complications of Diabetes | 8.5 | 161 | Citations (PDF) |
| 239 | Expression of Advanced Glycation End Products and Their Cellular Receptor RAGE in Diabetic Nephropathy and Nondiabetic Renal Disease | 0.4 | 434 | Citations (PDF) |
| 240 | N ε-(Carboxymethyl)Lysine Adducts of Proteins Are Ligands for Receptor for Advanced Glycation End Products That Activate Cell Signaling Pathways and Modulate Gene Expression | 2.2 | 833 | Citations (PDF) |
| 241 | Soluble cellular adhesion molecules in proliferative vitreoretinopathy and proliferative diabetic retinopathy | 2.1 | 44 | Citations (PDF) |
| 242 | Emerging therapeutic targets in diabetic vascular disease | 0.8 | 0 | Citations (PDF) |
| 243 | Chronic aminoguanidine attenuates renal dysfunction and injury in aging rats | 2.0 | 35 | Citations (PDF) |
| 244 | RAGE Mediates a Novel Proinflammatory Axis | 33.6 | 1,827 | Citations (PDF) |
| 245 | Cellular Cofactors for Amyloid β-Peptide-Induced Cell Stress | 3.4 | 37 | Citations (PDF) |
| 246 | Suppression of accelerated diabetic atherosclerosis by the soluble receptor for advanced glycation endproducts | 33.0 | 1,104 | Citations (PDF) |
| 247 | β2-microglobulin modified with advanced glycation end products modulates collagen synthesis by human fibroblasts | 5.3 | 87 | Citations (PDF) |
| 248 | Vasculopathy and insulin resistance in the JCR:LA-cp rat | 1.5 | 56 | Citations (PDF) |
| 249 | Enhanced Interaction of Advanced Glycation End Products With Their Cellular Receptor RAGE: Implications for the Pathogenesis of Accelerated Periodontal Disease in Diabetes | 6.8 | 100 | Citations (PDF) |
| 250 | Sp1-binding Elements in the Promoter of RAGE Are Essential for Amphoterin-mediated Gene Expression in Cultured Neuroblastoma Cells | 2.2 | 74 | Citations (PDF) |
| 251 | Vitamin E ameliorates enhanced renal lipid peroxidation and accumulation of F2-isoprostanes in aging kidneys | 2.4 | 41 | Citations (PDF) |
| 252 | A murine model of accelerated periodontal disease in diabetes | 3.4 | 81 | Citations (PDF) |
| 253 | Characterization and Functional Analysis of the Promoter of RAGE, the Receptor for Advanced Glycation End Products | 2.2 | 462 | Citations (PDF) |
| 254 | Pathogenesis of diabetic nephropathy: a radical approach | 0.8 | 68 | Citations (PDF) |
| 255 | Activation of the Receptor for Advanced Glycation End Products Triggers a p21 -dependent Mitogen-activated Protein Kinase Pathway Regulated by Oxidant Stress | 2.2 | 737 | Citations (PDF) |
| 256 | Recombinant Advanced Glycation End Product Receptor Pharmacokinetics in Normal and Diabetic Rats | 2.6 | 82 | Citations (PDF) |
| 257 | Endothelial-monocyte activating polypeptide ii, a novel anti-tumor cytokine that suppresses primary and metastatic tumor growth, and induces apoptosis in growing endothelial cells. | 6.6 | 0 | Citations (PDF) |
| 258 | The receptor for advanced glycation products (rage): Implications for the pathogenesis of diabetic complications. | 6.6 | 0 | Citations (PDF) |
| 259 | Receptors for advanced glycation end-products (AGE)—expression by endothelial cells in non-diabetic uraemic patients | 0.8 | 0 | Citations (PDF) |
| 260 | Elevated plasma levels of vascular cell adhesion molecule‐1 (VCAM‐1) in diabetic patients with microalbuminuria: a marker of vascular dysfunction and progressive vascular disease | 2.7 | 104 | Citations (PDF) |
| 261 | Advanced glycation endproducts (AGEs) induce oxidant stress in the gingiva: a potential mechanism underlying accelerated periodontal disease associated with diabetes | 3.4 | 261 | Citations (PDF) |
| 262 | RAGE and amyloid-β peptide neurotoxicity in Alzheimer's disease | 37.9 | 2,044 | Citations (PDF) |
| 263 | Receptors for advanced glycation end-products (AGE)--expression by endothelial cells in non-diabetic uraemic patients | 0.8 | 42 | Citations (PDF) |
| 264 | The receptor for advanced glycation end-products has a central role in mediating the effects of advanced glycation end-products on the development of vascular disease in diabetes mellitus | 0.8 | 75 | Citations (PDF) |
| 265 | Receptor-mediated endothelial cell dysfunction in diabetic vasculopathy. Soluble receptor for advanced glycation end products blocks hyperpermeability in diabetic rats. | 10.6 | 495 | Citations (PDF) |
| 266 | The receptor for advanced glycation end products (RAGE) is a central mediator of the interaction of AGE-beta2microglobulin with human mononuclear phagocytes via an oxidant-sensitive pathway. Implications for the pathogenesis of dialysis-related amyloidosis. | 10.6 | 275 | Citations (PDF) |
| 267 | The Receptor for Advanced Glycation End Products (RAGE) Is a Cellular Binding Site for Amphoterin | 2.2 | 1,090 | Citations (PDF) |
| 268 | Advanced glycation endproducts interacting with their endothelial receptor induce expression of vascular cell adhesion molecule-1 (VCAM-1) in cultured human endothelial cells and in mice. A potential mechanism for the accelerated vasculopathy of diabetes. | 10.6 | 829 | Citations (PDF) |
| 269 | Receptor for advanced glycation end products (AGEs) has a central role in vessel wall interactions and gene activation in response to circulating AGE proteins. | 7.5 | 315 | Citations (PDF) |
| 270 | Advanced glycation end products (AGEs) on the surface of diabetic erythrocytes bind to the vessel wall via a specific receptor inducing oxidant stress in the vasculature: a link between surface-associated AGEs and diabetic complications. | 7.5 | 337 | Citations (PDF) |
| 271 | Glycated tau protein in Alzheimer disease: a mechanism for induction of oxidant stress. | 7.5 | 585 | Citations (PDF) |
| 272 | Regulation of human mononuclear phagocyte migration by cell surface-binding proteins for advanced glycation end products. | 10.6 | 265 | Citations (PDF) |
| 273 | Resolution of fluorescence intensity decays of the two tryptophan residues in glutamine-binding protein from Escherichia coli using single tryptophan mutants | 2.2 | 27 | Citations (PDF) |
| 274 | Viral persistence during the developmental phase of Coxsackievirus B1-induced murine polymyositis | 3.6 | 38 | Citations (PDF) |
| 275 | Time-resolved studies define the nature of toxic IAPP intermediates, providing insight for anti-amyloidosis therapeutics | 0.7 | 159 | Citations (PDF) |
| 276 | ATVB
Journal Awards at Vascular Discovery | 6.0 | 0 | Citations (PDF) |