| 1 | Assays to Study IRE1 Activation and Signaling | 0.0 | 0 | Citations (PDF) |
| 2 | Stress-induced tyrosine phosphorylation of RtcB modulates IRE1 activity and signaling outputs | 2.7 | 10 | Citations (PDF) |
| 3 | A chosen STING with a PERKy trail | 10.5 | 2 | Citations (PDF) |
| 4 | Cell death regulation by MAMs: from molecular mechanisms to therapeutic implications in cardiovascular diseases | 8.5 | 33 | Citations (PDF) |
| 5 | Balancing energy and protein homeostasis at ER-mitochondria contact sites | 5.5 | 27 | Citations (PDF) |
| 6 | Mitochondria-associated niches in health and disease | 3.2 | 3 | Citations (PDF) |
| 7 | Simultaneous determination of intraluminal lysosomal calcium and pH by dextran-conjugated fluorescent dyes | 0.0 | 6 | Citations (PDF) |
| 8 | Disruption of Endoplasmic Reticulum Proteostasis in Age-Related Nervous System Disorders | 0.0 | 2 | Citations (PDF) |
| 9 | Therapeutic potential of insulin-like growth factor 2 in Huntington’s disease: controlling proteostasis to alleviate the load of misfolded protein | 3.9 | 2 | Citations (PDF) |
| 10 | Endoplasmic reticulum stress and unfolded protein response in cardiovascular diseases | 12.5 | 403 | Citations (PDF) |
| 11 | Protein disulfide isomerase ERp57 protects early muscle denervation in experimental ALS | 5.1 | 11 | Citations (PDF) |
| 12 | DEF8 and Autophagy-Associated Genes Are Altered in Mild Cognitive Impairment, Probable Alzheimer’s Disease Patients, and a Transgenic Model of the Disease | 2.7 | 7 | Citations (PDF) |
| 13 | Retinal Ganglion Cells Functional Changes in a Mouse Model of Alzheimer’s Disease Are Linked with Neurotransmitter Alterations | 2.7 | 5 | Citations (PDF) |
| 14 | A phenolic-rich extract from Ugni molinae berries reduces abnormal protein aggregation in a cellular model of Huntington’s disease | 2.5 | 11 | Citations (PDF) |
| 15 | Paradoxical implication of BAX/BAK in the persistence of tetraploid cells | 8.5 | 7 | Citations (PDF) |
| 16 | Proteostasis impairment and ER stress as a possible target to treat Parkinson's disease | 0.0 | 0 | Citations (PDF) |
| 17 | Inflammation-associated suppression of metabolic gene networks in acute and chronic liver disease | 6.0 | 28 | Citations (PDF) |
| 18 | Autophagy in hepatic adaptation to stress | 2.9 | 121 | Citations (PDF) |
| 19 | When Endoplasmic Reticulum Proteostasis Meets the DNA Damage Response | 15.3 | 55 | Citations (PDF) |
| 20 | The UPRosome – decoding novel biological outputs of IRE1α function | 3.2 | 36 | Citations (PDF) |
| 21 | Getting intimate: Lysosomes and ER rendezvous to control autophagy | 2.7 | 3 | Citations (PDF) |
| 22 | Caveolin-1 suppresses tumor formation through the inhibition of the unfolded protein response | 8.5 | 25 | Citations (PDF) |
| 23 | Genotoxic stress triggers the activation of IRE1α-dependent RNA decay to modulate the DNA damage response | 14.1 | 62 | Citations (PDF) |
| 24 | Mechanisms, regulation and functions of the unfolded protein response | 31.4 | 1,423 | Citations (PDF) |
| 25 | Emerging roles of the unfolded protein response (UPR) in the nervous system: A link with adaptive behavior to environmental stress? | 4.5 | 21 | Citations (PDF) |
| 26 | Insulin-like growth factor 2 (IGF2) protects against Huntington’s disease through the extracellular disposal of protein aggregates | 7.9 | 44 | Citations (PDF) |
| 27 | Acute Pannexin 1 Blockade Mitigates Early Synaptic Plasticity Defects in a Mouse Model of Alzheimer’s Disease | 3.5 | 21 | Citations (PDF) |
| 28 | Small Molecules to Improve ER Proteostasis in Disease | 14.8 | 59 | Citations (PDF) |
| 29 | Pharmacological targeting of the unfolded protein response for disease intervention | 7.3 | 191 | Citations (PDF) |
| 30 | The UFMylation System in Proteostasis and Beyond | 15.3 | 103 | Citations (PDF) |
| 31 | The p75NTR neurotrophin receptor is required to organize the mature neuromuscular synapse by regulating synaptic vesicle availability | 5.1 | 14 | Citations (PDF) |
| 32 | β-catenin aggregation in models of ALS motor neurons: GSK3β inhibition effect and neuronal differentiation | 5.2 | 16 | Citations (PDF) |
| 33 | Non-canonical function of IRE1α determines mitochondria-associated endoplasmic reticulum composition to control calcium transfer and bioenergetics | 10.5 | 182 | Citations (PDF) |
| 34 | Emerging Roles of the Endoplasmic Reticulum Associated Unfolded Protein Response in Cancer Cell Migration and Invasion | 4.0 | 63 | Citations (PDF) |
| 35 | Targeting of the unfolded protein response (UPR) as therapy for Parkinson's disease | 2.8 | 35 | Citations (PDF) |
| 36 | Brain organoids: a next step for humanized Alzheimer’s disease models? | 8.3 | 52 | Citations (PDF) |
| 37 | ER stress links aging to sporadic ALS | 2.5 | 11 | Citations (PDF) |
| 38 | Gene Therapy Strategies to Restore ER Proteostasis in Disease | 10.5 | 36 | Citations (PDF) |
| 39 | Interactome Screening Identifies the ER Luminal Chaperone Hsp47 as a Regulator of the Unfolded Protein Response Transducer IRE1α | 14.2 | 123 | Citations (PDF) |
| 40 | Targeting PERK signaling with the small molecule GSK2606414 prevents neurodegeneration in a model of Parkinson's disease | 5.2 | 124 | Citations (PDF) |
| 41 | Homeostatic interplay between FoxO proteins and ER proteostasis in cancer and other diseases | 14.2 | 8 | Citations (PDF) |
| 42 | Unraveling the role of motoneuron autophagy in ALS | 13.8 | 12 | Citations (PDF) |
| 43 | Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 | 13.7 | 4,465 | Citations (PDF) |
| 44 | Genome-wide circulating microRNA expression profiling reveals potential biomarkers for amyotrophic lateral sclerosis | 3.4 | 49 | Citations (PDF) |
| 45 | (off)Targeting UPR signaling: the race toward intervening ER proteostasis | 3.9 | 7 | Citations (PDF) |
| 46 | Dual
<scp>IRE</scp>
1
<scp>RN</scp>
ase functions dictate glioblastoma development | 7.2 | 126 | Citations (PDF) |
| 47 | A decay of the adaptive capacity of the unfolded protein response exacerbates Alzheimer's disease | 3.4 | 11 | Citations (PDF) |
| 48 | Calcium signaling at the endoplasmic reticulum: fine-tuning stress responses | 2.7 | 211 | Citations (PDF) |
| 49 | The Unfolded Protein Response and Cell Fate Control | 14.2 | 1,023 | Citations (PDF) |
| 50 | Emerging roles of <scp>ER</scp> stress in the etiology and pathogenesis of Alzheimer's disease | 5.5 | 187 | Citations (PDF) |
| 51 | Interplay Between the Unfolded Protein Response and Immune Function in the Development of Neurodegenerative Diseases | 5.0 | 35 | Citations (PDF) |
| 52 | Cyclosporine A binding to COX-2 reveals a novel signaling pathway that activates the IRE1α unfolded protein response sensor | 3.7 | 16 | Citations (PDF) |
| 53 | Endoplasmic reticulum stress signalling and the pathogenesis of non-alcoholic fatty liver disease | 2.9 | 635 | Citations (PDF) |
| 54 | ER Proteostasis Control of Neuronal Physiology and Synaptic Function | 13.4 | 89 | Citations (PDF) |
| 55 | Endoplasmic reticulum stress leads to accumulation of wild-type SOD1 aggregates associated with sporadic amyotrophic lateral sclerosis | 7.7 | 83 | Citations (PDF) |
| 56 | IRE1α governs cytoskeleton remodelling and cell migration
through a direct interaction with filamin A | 10.5 | 98 | Citations (PDF) |
| 57 | A new model to study cell-to-cell transfer of αSynuclein in vivo | 2.1 | 5 | Citations (PDF) |
| 58 | ER stress sensing mechanism: Putting off the brake on UPR transducers | 1.7 | 12 | Citations (PDF) |
| 59 | The ER proteostasis network in ALS: Determining the differential motoneuron vulnerability | 1.9 | 33 | Citations (PDF) |
| 60 | Autophagosomes cooperate in the degradation of intracellular C‐terminal fragments of the amyloid precursor protein
<i>via</i>
the MVB/lysosomal pathway | 0.7 | 39 | Citations (PDF) |
| 61 | The Endoplasmic Reticulum Chaperone GRP78/BiP Modulates Prion Propagation in vitro and in vivo | 3.7 | 74 | Citations (PDF) |
| 62 | Endoplasmic reticulum proteostasis in glioblastoma—From molecular mechanisms to therapeutic perspectives | 5.5 | 102 | Citations (PDF) |
| 63 | Disulfide cross-linked multimers of TDP-43 and spinal motoneuron loss in a TDP-43A315T ALS/FTD mouse model | 3.7 | 19 | Citations (PDF) |
| 64 | ER Stress and Neurodegenerative Disease: A Cause or Effect Relationship? | 0.0 | 28 | Citations (PDF) |
| 65 | Proteostasis disturbance in amyotrophic lateral sclerosis | 3.1 | 35 | Citations (PDF) |
| 66 | ER stress in neurodegenerative disease: from disease mechanisms to therapeutic interventions | 0.5 | 6 | Citations (PDF) |
| 67 | Drug repurposing to target proteostasis and prevent neurodegeneration: accelerating translational effortsBrain, 2017, 140, 1544-1547 | 8.9 | 17 | Citations (PDF) |
| 68 | Fine-Tuning ER Stress Signal Transducers to Treat Amyotrophic Lateral Sclerosis | 3.5 | 18 | Citations (PDF) |
| 69 | The Unfolded Protein Response: At the Intersection between Endoplasmic Reticulum Function and Mitochondrial Bioenergetics | 2.7 | 37 | Citations (PDF) |
| 70 | Fine-tuning PERK signaling to control cell fate under stress | 6.4 | 14 | Citations (PDF) |
| 71 | Desafíos en el diagnóstico de enfermedad de Creutzfeldt-Jakob: Caso clínico | 0.2 | 2 | Citations (PDF) |
| 72 | Commentary: XBP-1 Is a Cell-Nonautonomous Regulator of Stress Resistance and Longevity | 4.1 | 4 | Citations (PDF) |
| 73 | <scp>ALS</scp>‐linked protein disulfide isomerase variants cause motor dysfunction | 7.4 | 110 | Citations (PDF) |
| 74 | NFκB is a central regulator of protein quality control in response to protein aggregation stresses via autophagy modulation | 2.5 | 37 | Citations (PDF) |
| 75 | Injury to the nervous system: A look into the ER | 2.5 | 22 | Citations (PDF) |
| 76 | Endoplasmic Reticulum Stress and the Hallmarks of Cancer | 14.0 | 420 | Citations (PDF) |
| 77 | Gene therapy to target ER stress in brain diseases | 2.5 | 31 | Citations (PDF) |
| 78 | PREFACE: Divergent roles of ER stress in neurodegeneration and brain disorders | 2.5 | 1 | Citations (PDF) |
| 79 | Glucose Metabolism: A Sweet Relief of Alzheimer’s Disease | 3.9 | 68 | Citations (PDF) |
| 80 | Activation of the unfolded protein response promotes axonal regeneration after peripheral nerve injury | 3.7 | 72 | Citations (PDF) |
| 81 | Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) | 13.8 | 4,327 | Citations (PDF) |
| 82 | The intersection between growth factors, autophagy and ER stress: A new target to treat neurodegenerative diseases? | 2.5 | 40 | Citations (PDF) |
| 83 | ERp57 as a novel cellular factor controlling prion protein biosynthesis: Therapeutic potential of protein disulfide isomerases | 1.7 | 8 | Citations (PDF) |
| 84 | Mystery solved: Trehalose kickstarts autophagy by blocking glucose transport | 5.5 | 72 | Citations (PDF) |
| 85 | Targeting endoplasmic reticulum acetylation to restore proteostasis in Alzheimer’s diseaseBrain, 2016, 139, 650-652 | 8.9 | 7 | Citations (PDF) |
| 86 | ERp57 in neurodegeneration and regeneration | 3.9 | 15 | Citations (PDF) |
| 87 | Bursting the unfolded protein response accelerates axonal regeneration | 3.9 | 3 | Citations (PDF) |
| 88 | ER proteostasis disturbances in Parkinson's disease: novel insights | 4.1 | 25 | Citations (PDF) |
| 89 | Functional Role of the Disulfide Isomerase ERp57 in Axonal Regeneration | 2.5 | 39 | Citations (PDF) |
| 90 | When the Good Turns Bad 2015, , 259-272 | | 0 | Citations (PDF) |
| 91 | Meet Our Associate Editor: | 2.1 | 0 | Citations (PDF) |
| 92 | Theme Series – UPR in cancer | 14.2 | 8 | Citations (PDF) |
| 93 | ER stress signaling and neurodegeneration: At the intersection between Alzheimer's disease and Prion-related disorders | 2.6 | 27 | Citations (PDF) |
| 94 | Targeting the unfolded protein response for disease intervention | 3.9 | 55 | Citations (PDF) |
| 95 | Proteostasis control by the unfolded protein response | 10.5 | 567 | Citations (PDF) |
| 96 | ER proteostasis addiction in cancer biology: Novel concepts | 14.2 | 41 | Citations (PDF) |
| 97 | Endoplasmic Reticulum Stress–Activated Cell Reprogramming in Oncogenesis | 26.4 | 295 | Citations (PDF) |
| 98 | <scp>RNA</scp>
metabolism: putting the brake on the
<scp>UPR</scp> | 5.3 | 4 | Citations (PDF) |
| 99 | Dual Role of Autophagy in Neurodegenerative Diseases: The Case of Amyotrophic Lateral Sclerosis | 0.0 | 2 | Citations (PDF) |
| 100 | The Protein-disulfide Isomerase ERp57 Regulates the Steady-state Levels of the Prion Protein | 2.3 | 49 | Citations (PDF) |
| 101 | PERK regulated miR-424(322)-503 cluster fine-tunes activation of IRE1 and ATF6 during Unfolded Protein Response | 3.7 | 35 | Citations (PDF) |
| 102 | Gene therapy in Parkinson′s disease: targeting the endoplasmic reticulum proteostasis network | 3.9 | 6 | Citations (PDF) |
| 103 | Memory loss in Alzheimer's disease: are the alterations in the UPR network involved in the cognitive impairment? | 4.1 | 33 | Citations (PDF) |
| 104 | A new method to measure autophagy flux in the nervous system | 13.8 | 24 | Citations (PDF) |
| 105 | Modeling UPR adaptive responses | 7.3 | 3 | Citations (PDF) |
| 106 | <scp>RESET</scp>
ing
<scp>ER</scp>
proteostasis: selective stress pathway hidden in the secretory route | 7.4 | 6 | Citations (PDF) |
| 107 | Cellular Mechanisms of Endoplasmic Reticulum Stress Signaling in Health and Disease. 1. An overview | 4.4 | 139 | Citations (PDF) |
| 108 | Autophagy meets fused in sarcoma-positive stress granules | 3.4 | 13 | Citations (PDF) |
| 109 | Addicted to secrete – novel concepts and targets in cancer therapy | 10.0 | 72 | Citations (PDF) |
| 110 | Pathogenic role of BECN1/Beclin 1 in the development of amyotrophic lateral sclerosis | 13.8 | 90 | Citations (PDF) |
| 111 | Targeting autophagy in neurodegenerative diseases | 14.8 | 129 | Citations (PDF) |
| 112 | The transcription factor CHOP, a central component of the transcriptional regulatory network induced upon CCl4 intoxication in mouse liver, is not a critical mediator of hepatotoxicity | 6.0 | 38 | Citations (PDF) |
| 113 | Interplay Between the Oxidoreductase PDIA6 and microRNA-322 Controls the Response to Disrupted Endoplasmic Reticulum Calcium Homeostasis | 5.5 | 89 | Citations (PDF) |
| 114 | Targeting the unfolded protein response in disease | 39.3 | 747 | Citations (PDF) |
| 115 | Herp depletion protects from protein aggregation by up-regulating autophagy | 3.6 | 31 | Citations (PDF) |
| 116 | A failure in energy metabolism and antioxidant uptake precede symptoms of Huntington’s disease in mice | 14.1 | 95 | Citations (PDF) |
| 117 | Bax Inhibitor-1-mediated Ca2+ leak is decreased by cytosolic acidosis | 2.7 | 27 | Citations (PDF) |
| 118 | An ERcentric view of Parkinson's disease | 10.0 | 160 | Citations (PDF) |
| 119 | The unfolded protein response in Alzheimer’s disease | 8.5 | 94 | Citations (PDF) |
| 120 | The biological meaning of the UPR | 31.4 | 25 | Citations (PDF) |
| 121 | Unspliced XBP1 controls autophagy through FoxO1 | 8.2 | 25 | Citations (PDF) |
| 122 | ER Dysfunction and Protein Folding Stress in ALS | 2.3 | 79 | Citations (PDF) |
| 123 | Trehalose delays the progression of amyotrophic lateral sclerosis by enhancing autophagy in motoneurons | 13.8 | 297 | Citations (PDF) |
| 124 | Role of the unfolded protein response in organ physiology: Lessons from mouse models | 3.0 | 44 | Citations (PDF) |
| 125 | Functional Contribution of the Transcription Factor ATF4 to the Pathogenesis of Amyotrophic Lateral Sclerosis | 2.5 | 80 | Citations (PDF) |
| 126 | Crosstalk between the UPR and autophagy pathway contributes to handling cellular stress in neurodegenerative disease | 13.8 | 48 | Citations (PDF) |
| 127 | Bax channel inhibitors prevent mitochondrion-mediated apoptosis and protect neurons in a model of global brain ischemia. | 2.3 | 1 | Citations (PDF) |
| 128 | Protein disulfide isomerases in neurodegeneration: From disease mechanisms to biomedical applications | 2.8 | 88 | Citations (PDF) |
| 129 | AAV-mediated delivery of the transcription factor XBP1s into the striatum reduces mutant Huntingtin aggregation in a mouse model of Huntington’s disease | 2.1 | 67 | Citations (PDF) |
| 130 | Targeting the UPR transcription factor XBP1 protects against Huntington's disease through the regulation of FoxO1 and autophagy | 3.1 | 246 | Citations (PDF) |
| 131 | Autophagy impairment: a crossroad between neurodegeneration and tauopathies | 4.0 | 25 | Citations (PDF) |
| 132 | Guidelines for the use and interpretation of assays for monitoring autophagy | 13.8 | 2,928 | Citations (PDF) |
| 133 | Cell‐nonautonomous control of the UPR | 5.3 | 4 | Citations (PDF) |
| 134 | Hormesis | 13.8 | 64 | Citations (PDF) |
| 135 | Altered Prion Protein Expression Pattern in CSF as a Biomarker for Creutzfeldt-Jakob Disease | 2.5 | 33 | Citations (PDF) |
| 136 | A BAX/BAK and Cyclophilin D-Independent Intrinsic Apoptosis Pathway | 2.5 | 34 | Citations (PDF) |
| 137 | The unfolded protein response: controlling cell fate decisions under ER stress and beyond | 31.4 | 3,069 | Citations (PDF) |
| 138 | Astrocytic αVβ3 Integrin Inhibits Neurite Outgrowth and Promotes Retraction of Neuronal Processes by Clustering Thy-1 | 2.5 | 57 | Citations (PDF) |
| 139 | Amyloid β-Peptide Oligomers Stimulate RyR-Mediated Ca<sup>2+</sup> Release Inducing Mitochondrial Fragmentation in Hippocampal Neurons and Prevent RyR-Mediated Dendritic Spine Remodeling Produced by BDNF | 6.4 | 117 | Citations (PDF) |
| 140 | Modulating stress responses by the UPRosome: A matter of life and death | 8.1 | 218 | Citations (PDF) |
| 141 | Oxidative stress activates the c-Abl/p73 proapoptotic pathway in Niemann-Pick type C neurons | 5.2 | 57 | Citations (PDF) |
| 142 | Integrating stress signals at the endoplasmic reticulum: The BCL-2 protein family rheostat | 3.6 | 136 | Citations (PDF) |
| 143 | Protein folding stress in neurodegenerative diseases: a glimpse into the ER | 4.2 | 193 | Citations (PDF) |
| 144 | Protein homeostasis networks in physiology and disease | 4.2 | 33 | Citations (PDF) |
| 145 | Abnormal calcium homeostasis and protein folding stress at the ER | 1.0 | 30 | Citations (PDF) |
| 146 | Targeting autophagy in ALS: A complex mission | 13.8 | 31 | Citations (PDF) |
| 147 | Axonal Degeneration Is Mediated by the Mitochondrial Permeability Transition Pore | 3.7 | 176 | Citations (PDF) |
| 148 | BAX inhibitor-1 regulates autophagy by controlling the IRE1α branch of the unfolded protein response | 7.4 | 104 | Citations (PDF) |
| 149 | Prion Protein Misfolding Affects Calcium Homeostasis and Sensitizes Cells to Endoplasmic Reticulum Stress | 2.5 | 70 | Citations (PDF) |
| 150 | HSP72 Protects Cells from ER Stress-induced Apoptosis via Enhancement of IRE1α-XBP1 Signaling through a Physical Interaction | 5.2 | 201 | Citations (PDF) |
| 151 | Amyotrophic Lateral Sclerosis Pathogenesis: A Journey Through the Secretory Pathway | 6.4 | 51 | Citations (PDF) |
| 152 | Alternative Functions of the BCL-2 Protein Family at the Endoplasmic Reticulum | 0.0 | 11 | Citations (PDF) |
| 153 | XBP-1 deficiency in the nervous system reveals a homeostatic switch to activate autophagy | 13.8 | 33 | Citations (PDF) |
| 154 | Turning off the unfolded protein response: An interplay between the apoptosis machinery and ER stress signaling | 3.2 | 21 | Citations (PDF) |
| 155 | The UPR as a survival factor of cancer cells: More than folding proteins? | 0.6 | 16 | Citations (PDF) |
| 156 | Fine-Tuning of the Unfolded Protein Response: Assembling the IRE1α Interactome | 14.2 | 350 | Citations (PDF) |
| 157 | The daily job of night killers: alternative roles of the BCL-2 family in organelle physiology | 15.3 | 78 | Citations (PDF) |
| 158 | XBP-1 and the UPRosome: Mastering Secretory Cell Function | 0.1 | 14 | Citations (PDF) |
| 159 | Editorial [ Apoptosis, Necrosis and Autophagy: From Mechanisms to Biomedical Applications (Part-I) Guest Editor: Claudio Hetz ] | 2.1 | 5 | Citations (PDF) |
| 160 | The Stress Rheostat: An Interplay Between the Unfolded Protein Response (UPR) and Autophagy in Neurodegeneration | 2.1 | 116 | Citations (PDF) |
| 161 | Perturbation of Endoplasmic Reticulum Homeostasis Facilitates Prion Replication | 2.3 | 57 | Citations (PDF) |
| 162 | Prion Pathogenesis is Independent of Caspase-12 | 1.7 | 41 | Citations (PDF) |
| 163 | Editorial [Hot Topic: Emerging Roles of the Unfolded Protein Response Signaling in Physiology and Disease (Executive Editor: Claudio A. Hetz and Claudio Soto )] | 2.1 | 14 | Citations (PDF) |
| 164 | Protein Misfolding 2005, , 213-227 | | 0 | Citations (PDF) |
| 165 | Signaling Pathways Controling Prion Neurotoxicity: Role of Endoplasmic Reticulum Stress-Mediated Apoptosis 2005, , 319-344 | | 1 | Citations (PDF) |
| 166 | Bax Channel Inhibitors Prevent Mitochondrion-mediated Apoptosis and Protect Neurons in a Model of Global Brain Ischemia | 2.3 | 148 | Citations (PDF) |
| 167 | Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia | 7.7 | 705 | Citations (PDF) |
| 168 | In Vitro Generation of Infectious Scrapie Prions | 35.1 | 676 | Citations (PDF) |
| 169 | Prion Replication Alters the Distribution of Synaptophysin and Caveolin 1 in Neuronal Lipid Rafts | 3.6 | 47 | Citations (PDF) |
| 170 | Molecular Mechanisms of Neurotoxicity of Pathological Prion Protein | 2.1 | 41 | Citations (PDF) |
| 171 | Caspase-12 and endoplasmic reticulum stress mediate neurotoxicity of pathological prion protein | 7.4 | 333 | Citations (PDF) |
| 172 | Is loss of function of the prion protein the cause of prion disorders? | 10.0 | 61 | Citations (PDF) |
| 173 | Microcin E492, a channel-forming bacteriocin from Klebsiella pneumoniae, induces apoptosis in some human cell lines | 7.7 | 138 | Citations (PDF) |
| 174 | Nonselective cation channels as effectors of free radical–induced rat liver cell necrosis | 11.6 | 56 | Citations (PDF) |
| 175 | Structure, organization and characterization of the gene cluster involved in the production of microcin E492, a channel‐forming bacteriocin | 2.7 | 66 | Citations (PDF) |
