| 1 | An autopsy case of type A FTLD‐TDP with a GRN mutation presenting with the logopenic variant of primary progressive aphasia at onset and with corticobasal syndrome subsequently | 0.7 | 1 | Citations (PDF) |
| 2 | Motor involvement in frontotemporal lobar degeneration with TAR DNA‐binding protein of 43 kDa type C | 0.7 | 7 | Citations (PDF) |
| 3 | Atypical TDP‐43 proteinopathy clinically presenting with progressive nonfluent aphasia: A case report | 0.7 | 0 | Citations (PDF) |
| 4 | Ultrastructures of α-Synuclein Filaments in Synucleinopathy Brains and Experimental Models | 1.0 | 9 | Citations (PDF) |
| 5 | The new missense G376V-TDP-43 variant induces late-onset distal myopathy but not amyotrophic lateral sclerosisBrain, 2024, 147, 1768-1783 | 5.9 | 16 | Citations (PDF) |
| 6 | Corticobasal degeneration with visual hallucination as an initial symptom: A case report | 0.7 | 3 | Citations (PDF) |
| 7 | Clinicopathological study of dementia with grains presenting with parkinsonism compared with a typical case | 0.7 | 5 | Citations (PDF) |
| 8 | Selection of lansoprazole from an FDA-approved drug library to inhibit the Alzheimer’s disease seed-dependent formation of tau aggregates | 2.7 | 7 | Citations (PDF) |
| 9 | Tau filaments with the chronic traumatic encephalopathy fold in a case of vacuolar tauopathy with VCP mutation D395G | 5.8 | 23 | Citations (PDF) |
| 10 | Long-standing preservation of levodopa response in progressive supranuclear palsy | 1.2 | 1 | Citations (PDF) |
| 11 | Heteromeric amyloid filaments of ANXA11 and TDP-43 in FTLD-TDP type C | 30.7 | 96 | Citations (PDF) |
| 12 | Analysis and comparison of post-translational modifications of α-synuclein filaments in multiple system atrophy and dementia with Lewy bodies | 2.7 | 10 | Citations (PDF) |
| 13 | An autopsy case of progressive supranuclear palsy treated with monoclonal antibody against tau | 0.7 | 5 | Citations (PDF) |
| 14 | Neuropathology of spinocerebellar ataxia type 8: Common features and unique tauopathy | 0.7 | 16 | Citations (PDF) |
| 15 | Identical tau filaments in subacute sclerosing panencephalitis and chronic traumatic encephalopathy | 3.3 | 41 | Citations (PDF) |
| 16 | Tunnelling nanotubes between neuronal and microglial cells allow bi-directional transfer of α-Synuclein and mitochondria | 6.6 | 183 | Citations (PDF) |
| 17 | TDP-43 forms amyloid filaments with a distinct fold in type A FTLD-TDP | 30.7 | 163 | Citations (PDF) |
| 18 | Distinct tau folds initiate templated seeding and alter the post-translational modification profileBrain, 2023, 146, 4988-4999 | 5.9 | 15 | Citations (PDF) |
| 19 | Dysregulation of the progranulin-driven autophagy-lysosomal pathway mediates secretion of the nuclear protein TDP-43 | 1.3 | 21 | Citations (PDF) |
| 20 | Clinical course of pathologically confirmed corticobasal degeneration and corticobasal syndrome | 2.0 | 32 | Citations (PDF) |
| 21 | Development of a novel tau propagation mouse model endogenously expressing 3 and 4 repeat tau isoformsBrain, 2022, 145, 349-361 | 5.9 | 28 | Citations (PDF) |
| 22 | Phosphorylation of endogenous α-synuclein induced by extracellular seeds initiates at the pre-synaptic region and spreads to the cell body | 2.7 | 42 | Citations (PDF) |
| 23 | An autopsy case of Alzheimer's disease with amygdala‐predominant Lewy pathology presenting with frontotemporal dementia‐like psychiatric symptoms | 0.7 | 2 | Citations (PDF) |
| 24 | Age-dependent formation of TMEM106B amyloid filaments in human brains | 30.7 | 191 | Citations (PDF) |
| 25 | Motor neuron TDP-43 proteinopathy in progressive supranuclear palsy and corticobasal degenerationBrain, 2022, 145, 2769-2784 | 5.9 | 36 | Citations (PDF) |
| 26 | Independent distribution between tauopathy secondary to subacute sclerotic panencephalitis and measles virus: An immunohistochemical analysis in autopsy cases including cases treated with aggressive antiviral therapies | 3.0 | 24 | Citations (PDF) |
| 27 | Ultrastructural and biochemical classification of pathogenic tau, α-synuclein and TDP-43 | 5.8 | 59 | Citations (PDF) |
| 28 | Frontotemporal Lobar Degeneration With Unclassifiable 4-Repeat Tauopathy Mimicking Globular Glial Tauopathy | 1.4 | 1 | Citations (PDF) |
| 29 | Structures of tau and α-synuclein filaments from brains of patients with neurodegenerative diseases | 3.4 | 4 | Citations (PDF) |
| 30 | Casein kinase 1δ/ε phosphorylates fused in sarcoma (FUS) and ameliorates FUS-mediated neurodegeneration | 1.3 | 10 | Citations (PDF) |
| 31 | An autopsy case of progressive supranuclear palsy. Pallido‐nigro‐luysian type with argyrophilic grains clinically presenting with personality and behavioral changes | 0.7 | 4 | Citations (PDF) |
| 32 | Structures of α-synuclein filaments from human brains with Lewy pathology | 30.7 | 489 | Citations (PDF) |
| 33 | An autopsy case of corticobasal syndrome due to asymmetric degeneration of the motor cortex and substantia nigra with TDP‐43 proteinopathy, associated with Alzheimer's disease pathology | 0.7 | 5 | Citations (PDF) |
| 34 | Seeded assembly
in vitro
does not replicate the structures of α‐synuclein filaments from multiple system atrophy | 1.5 | 188 | Citations (PDF) |
| 35 | Progression of phosphorylated α‐synuclein in Macaca fuscata | 3.0 | 10 | Citations (PDF) |
| 36 | Human tauopathy-derived tau strains determine the substrates recruited for templated amplificationBrain, 2021, 144, 2333-2348 | 5.9 | 38 | Citations (PDF) |
| 37 | An autopsy case of corticobasal degeneration with inferior olivary hypertrophy | 0.7 | 2 | Citations (PDF) |
| 38 | Amyotrophic lateral sclerosis with speech apraxia, predominant upper motor neuron signs, and prominent iron accumulation in the frontal operculum and precentral gyrus | 0.7 | 11 | Citations (PDF) |
| 39 | The hot cross bun sign in corticobasal degeneration | 0.7 | 9 | Citations (PDF) |
| 40 | α-Synuclein fibrils subvert lysosome structure and function for the propagation of protein misfolding between cells through tunneling nanotubes | 3.1 | 122 | Citations (PDF) |
| 41 | Structure-based classification of tauopathies | 30.7 | 828 | Citations (PDF) |
| 42 | Distinct phosphorylation profiles of tau in brains of patients with different tauopathies | 2.4 | 30 | Citations (PDF) |
| 43 | An immigrant family with Kii amyotrophic lateral sclerosis/parkinsonism–dementia complex | 1.1 | 4 | Citations (PDF) |
| 44 | Structure of pathological TDP-43 filaments from ALS with FTLD | 30.7 | 313 | Citations (PDF) |
| 45 | Structurally Distinct α‐Synuclein Fibrils Induce Robust Parkinsonian Pathology | 3.2 | 32 | Citations (PDF) |
| 46 | Globular glial tauopathy Type I presenting with behavioral variant frontotemporal dementia | 0.7 | 11 | Citations (PDF) |
| 47 | Asparagine residue 368 is involved in Alzheimer's disease tau strain–specific aggregation | 1.3 | 15 | Citations (PDF) |
| 48 | Comparison of Common and Disease-Specific Post-translational Modifications of Pathological Tau Associated With a Wide Range of Tauopathies | 2.0 | 77 | Citations (PDF) |
| 49 | Structures of α-synuclein filaments from multiple system atrophy | 30.7 | 792 | Citations (PDF) |
| 50 | Novel tau filament fold in corticobasal degeneration | 30.7 | 538 | Citations (PDF) |
| 51 | Dextran sulphate-induced tau assemblies cause endogenous tau aggregation and propagation in wild-type mice | 2.0 | 16 | Citations (PDF) |
| 52 | Factors associated with development and distribution of granular/fuzzy astrocytes in neurodegenerative diseases | 3.0 | 15 | Citations (PDF) |
| 53 | Prion-like Propagation Model of Tau | 0.2 | 0 | Citations (PDF) |
| 54 | LATE to the PART-yBrain, 2019, 142, e47-e47 | 5.9 | 59 | Citations (PDF) |
| 55 | Prion-like Propagation of Pathological α-Synuclein <i>in Vivo</i> | 0.2 | 3 | Citations (PDF) |
| 56 | Tau progression in single severe frontal traumatic brain injury in human brains | 1.2 | 19 | Citations (PDF) |
| 57 | Mutations in CHCHD2 cause α-synuclein aggregation | 2.1 | 75 | Citations (PDF) |
| 58 | Human NPCs can degrade α–syn fibrils and transfer them preferentially in a cell contact-dependent manner possibly through TNT-like structures | 3.5 | 33 | Citations (PDF) |
| 59 | The basis of clinicopathological heterogeneity in TDP-43 proteinopathy | 5.8 | 125 | Citations (PDF) |
| 60 | Tau isoform expression and phosphorylation in marmoset brains | 1.3 | 43 | Citations (PDF) |
| 61 | An autopsy case of globular glial tauopathy presenting with clinical features of motor neuron disease with dementia and iron deposition in the motor cortex | 0.7 | 16 | Citations (PDF) |
| 62 | Isoform-independent and -dependent phosphorylation of microtubule-associated protein tau in mouse brain during postnatal development | 1.3 | 46 | Citations (PDF) |
| 63 | Progranulin haploinsufficiency reduces amyloid beta deposition in Alzheimer’s disease model mice | 12.5 | 21 | Citations (PDF) |
| 64 | TDP-43 Prions | 2.9 | 42 | Citations (PDF) |
| 65 | Unusual tau pathology of the cerebellum in patients with amyotrophic lateral sclerosis/parkinsonism–dementia complex from the Kii Peninsula, Japan | 3.0 | 16 | Citations (PDF) |
| 66 | N-Methyl-D-Aspartate Receptor Link to the MAP Kinase Pathway in Cortical and Hippocampal Neurons and Microglia Is Dependent on Calcium Sensors and Is Blocked by α-Synuclein, Tau, and Phospho-Tau in Non-transgenic and Transgenic APPSw,Ind Mice | 2.4 | 31 | Citations (PDF) |
| 67 | The effect of truncation on prion-like properties of α-synuclein | 1.3 | 84 | Citations (PDF) |
| 68 | Reconsideration of Amyloid Hypothesis and Tau Hypothesis in Alzheimer's Disease | 2.0 | 781 | Citations (PDF) |
| 69 | Potent prion-like behaviors of pathogenic α-synuclein and evaluation of inactivation methods | 3.3 | 106 | Citations (PDF) |
| 70 | C9ORF72 dipeptide repeat poly-GA inclusions promote intracellular aggregation of phosphorylated TDP-43 | 2.1 | 48 | Citations (PDF) |
| 71 | Tau-related dysfunction of BRCA1 lead to reduced neuronal plasticity in Alzheimer’s disease (P2.191) | 0.7 | 0 | Citations (PDF) |
| 72 | Progranulin regulates lysosomal function and biogenesis through acidification of lysosomes | 2.1 | 131 | Citations (PDF) |
| 73 | Propagation of pathological α-synuclein in marmoset brain | 3.3 | 178 | Citations (PDF) |
| 74 | Accumulation of multiple neurodegenerative disease-related proteins in familial frontotemporal lobar degeneration associated with granulin mutation | 2.7 | 40 | Citations (PDF) |
| 75 | Prion-like mechanisms and potential therapeutic targets in neurodegenerative disorders 2017, 172, 22-33 | | 63 | Citations (PDF) |
| 76 | Wild-Type Monomeric α-Synuclein Can Impair Vesicle Endocytosis and Synaptic Fidelity via Tubulin Polymerization at the Calyx of Held | 2.3 | 64 | Citations (PDF) |
| 77 | Desulfation of Heparan Sulfate by Sulf1 and Sulf2 Is Required for Corticospinal Tract Formation | 2.7 | 30 | Citations (PDF) |
| 78 | Following the fate of endocytosed fibrils | 1.3 | 1 | Citations (PDF) |
| 79 | Molecular Mechanisms in the Pathogenesis of Alzheimer’s disease and Tauopathies-Prion-Like Seeded Aggregation and Phosphorylation | 3.1 | 69 | Citations (PDF) |
| 80 | The Effect of Fragmented Pathogenic α-Synuclein Seeds on Prion-like Propagation | 1.3 | 120 | Citations (PDF) |
| 81 | Mass spectrometric analysis of accumulated TDP-43 in amyotrophic lateral sclerosis brains | 2.7 | 166 | Citations (PDF) |
| 82 | Quantitative and combinatory determination of in situ phosphorylation of tau and its FTDP-17 mutants | 2.7 | 25 | Citations (PDF) |
| 83 | α-Synuclein Fibrils Exhibit Gain of Toxic Function, Promoting Tau Aggregation and Inhibiting Microtubule Assembly | 1.3 | 84 | Citations (PDF) |
| 84 | Pathological tau deposition in Motor Neurone Disease and frontotemporal lobar degeneration associated with TDP-43 proteinopathy | 3.3 | 38 | Citations (PDF) |
| 85 | Gain-of-function profilin 1 mutations linked to familial amyotrophic lateral sclerosis cause seed-dependent intracellular TDP-43 aggregation | 2.1 | 65 | Citations (PDF) |
| 86 | Templated Aggregation of TAR DNA-binding Protein of 43 kDa (TDP-43) by Seeding with TDP-43 Peptide Fibrils | 1.3 | 99 | Citations (PDF) |
| 87 | α-Synuclein: Experimental Pathology | 2.9 | 30 | Citations (PDF) |
| 88 | The Abundance of Nonphosphorylated Tau in Mouse and Human Tauopathy Brains Revealed by the Use of Phos-Tag Method | 2.8 | 22 | Citations (PDF) |
| 89 | ALS patients with ability to communicate after long-term mechanical ventilation have confined degeneration to the motor neuron system | 1.2 | 9 | Citations (PDF) |
| 90 | The Relationship Between Development of Neuronal and Astrocytic Tau Pathologies in Subcortical Nuclei and Progression of Argyrophilic Grain Disease | 3.0 | 25 | Citations (PDF) |
| 91 | Extracellular association of APP and tau fibrils induces intracellular aggregate formation of tau | 5.8 | 79 | Citations (PDF) |
| 92 | Progranulin Reduction Is Associated With Increased Tau Phosphorylation in P301L Tau Transgenic Mice | 1.4 | 59 | Citations (PDF) |
| 93 | The twenty-four KDa C-terminal tau fragment increases with aging in tauopathy mice: implications of prion-like properties | 2.1 | 61 | Citations (PDF) |
| 94 | Biochemical classification of tauopathies by immunoblot, protein sequence and mass spectrometric analyses of sarkosyl-insoluble and trypsin-resistant tau | 5.8 | 198 | Citations (PDF) |
| 95 | Differential diagnosis of amyotrophic lateral sclerosis from Guillain–Barré syndrome by quantitative determination of TDP-43 in cerebrospinal fluid | 1.2 | 33 | Citations (PDF) |
| 96 | Cyclin-dependent kinase 5 phosphorylates and induces the degradation of ataxin-2 | 1.3 | 17 | Citations (PDF) |
| 97 | Distinct pathways leading to TDP-43-induced cellular dysfunctions | 2.1 | 36 | Citations (PDF) |
| 98 | Pathological alpha-synuclein propagates through neural networks | 3.3 | 233 | Citations (PDF) |
| 99 | Tau accumulation in the nucleus accumbens in tangle-predominant dementia | 3.3 | 38 | Citations (PDF) |
| 100 | Pathological alpha-synuclein propagates through neural networks | 3.3 | 17 | Citations (PDF) |
| 101 | Prion-like Properties of Pathological TDP-43 Aggregates from Diseased Brains | 4.4 | 500 | Citations (PDF) |
| 102 | A longitudinal study on α-synuclein in blood plasma as a biomarker for Parkinson's disease | 2.7 | 187 | Citations (PDF) |
| 103 | Extensive deamidation at asparagine residue 279 accounts for weak immunoreactivity of tau with RD4 antibody in Alzheimer’s disease brain | 3.3 | 70 | Citations (PDF) |
| 104 | Prion-like spreading of pathological α-synuclein in brainBrain, 2013, 136, 1128-1138 | 5.9 | 830 | Citations (PDF) |
| 105 | Isomerase Pin1 Stimulates Dephosphorylation of Tau Protein at Cyclin-dependent Kinase (Cdk5)-dependent Alzheimer Phosphorylation Sites | 1.3 | 72 | Citations (PDF) |
| 106 | 3R and 4R tau isoforms in paired helical filaments in Alzheimer’s disease | 5.8 | 31 | Citations (PDF) |
| 107 | Regulation of Mitochondrial Transport and Inter-Microtubule Spacing by Tau Phosphorylation at the Sites Hyperphosphorylated in Alzheimer's Disease | 2.3 | 191 | Citations (PDF) |
| 108 | Molecular analysis and biochemical classification of TDP-43 proteinopathyBrain, 2012, 135, 3380-3391 | 5.9 | 112 | Citations (PDF) |
| 109 | Prolonged nitric oxide treatment induces tau aggregation in SH-SY5Y cells | 1.3 | 12 | Citations (PDF) |
| 110 | Epitope mapping of antibodies against TDP-43 and detection of protease-resistant fragments of pathological TDP-43 in amyotrophic lateral sclerosis and frontotemporal lobar degeneration | 1.5 | 35 | Citations (PDF) |
| 111 | Oxidative stress induced by glutathione depletion reproduces pathological modifications of TDP-43 linked to TDP-43 proteinopathies | 3.5 | 102 | Citations (PDF) |
| 112 | Methylene Blue Reduced Abnormal Tau Accumulation in P301L Tau Transgenic Mice | 1.5 | 89 | Citations (PDF) |
| 113 | "Protein cancers" hypothesis for neurodegenerative diseases | 0.1 | 1 | Citations (PDF) |
| 114 | Molecular neuropathology of neurodegeneration by TDP-43 | 0.1 | 0 | Citations (PDF) |
| 115 | Molecular Dissection of TDP-43 Proteinopathies | 1.4 | 51 | Citations (PDF) |
| 116 | Molecular dissection of TDP-43 in ALS and FTLD | 0.1 | 0 | Citations (PDF) |
| 117 | Phosphorylated TDP-43 pathology and hippocampal sclerosis in progressive supranuclear palsy | 5.8 | 117 | Citations (PDF) |
| 118 | Effect of topographical distribution of α-synuclein pathology on TDP-43 accumulation in Lewy body disease | 5.8 | 42 | Citations (PDF) |
| 119 | Phosphorylated and cleaved TDP-43 in ALS, FTLD and other neurodegenerative disorders and in cellular models of TDP-43 proteinopathy | 0.7 | 124 | Citations (PDF) |
| 120 | Occurrence of basophilic inclusions and FUS-immunoreactive neuronal and glial inclusions in a case of familial amyotrophic lateral sclerosis | 1.2 | 22 | Citations (PDF) |
| 121 | Characterization of Inhibitor-Bound α-Synuclein Dimer: Role of α-Synuclein N-Terminal Region in Dimerization and Inhibitor Binding | 2.9 | 25 | Citations (PDF) |
| 122 | Seeded Aggregation and Toxicity of α-Synuclein and Tau | 1.3 | 327 | Citations (PDF) |
| 123 | Effect of Pin1 or Microtubule Binding on Dephosphorylation of FTDP-17 Mutant Tau | 1.3 | 22 | Citations (PDF) |
| 124 | Truncation and pathogenic mutations facilitate the formation of intracellular aggregates of TDP-43 | 2.1 | 288 | Citations (PDF) |
| 125 | Conversion of Wild-type α-Synuclein into Mutant-type Fibrils and Its Propagation in the Presence of A30P Mutant | 1.3 | 99 | Citations (PDF) |
| 126 | Phosphorylated TDP-43 in Alzheimer’s disease and dementia with Lewy bodies | 5.8 | 333 | Citations (PDF) |
| 127 | TDP-43 pathology in familial British dementia | 5.8 | 24 | Citations (PDF) |
| 128 | Plasma phosphorylated-TDP-43 protein levels correlate with brain pathology in frontotemporal lobar degeneration | 5.8 | 93 | Citations (PDF) |
| 129 | A Cellular Model To Monitor Proteasome Dysfunction by α-Synuclein | 1.5 | 58 | Citations (PDF) |
| 130 | Identification of casein kinase-1 phosphorylation sites on TDP-43 | 1.5 | 118 | Citations (PDF) |
| 131 | The TDP-43 proteinopathies, toward understanding of the molecular pathogenesis | 0.1 | 2 | Citations (PDF) |
| 132 | Phosphorylated TDP‐43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis | 4.6 | 764 | Citations (PDF) |
| 133 | Relationship of phosphorylated α-synuclein and tau accumulation to Aβ deposition in the cerebral cortex of dementia with Lewy bodies | 2.9 | 96 | Citations (PDF) |
| 134 | Colocalization of Transactivation-Responsive DNA-Binding Protein 43 and Huntingtin in Inclusions of Huntington Disease | 1.4 | 261 | Citations (PDF) |
| 135 | The significance of the TDP-43 deposition in FTLD-U and ALS | 0.1 | 1 | Citations (PDF) |
| 136 | Accumulation of phosphorylated TDP-43 in brains of patients with argyrophilic grain disease | 5.8 | 100 | Citations (PDF) |
| 137 | TDP-43 is deposited in the Guam parkinsonism-dementia complex brainsBrain, 2007, 130, 1386-1394 | 5.9 | 233 | Citations (PDF) |
| 138 | Fibrillogenic Nuclei Composed of P301L Mutant Tau Induce Elongation of P301L Tau but Not Wild-type Tau* | 1.3 | 81 | Citations (PDF) |
| 139 | Small Molecule Inhibitors of α-Synuclein Filament Assembly† | 1.5 | 375 | Citations (PDF) |
| 140 | Cysteine misincorporation in bacterially expressed human α-synuclein | 1.8 | 81 | Citations (PDF) |
| 141 | TDP-43 is a component of ubiquitin-positive tau-negative inclusions in frontotemporal lobar degeneration and amyotrophic lateral sclerosis | 1.5 | 2,670 | Citations (PDF) |
| 142 | Corticobasal degeneration with focal, massive tau accumulation in the subcortical white matter astrocytes | 5.8 | 26 | Citations (PDF) |
| 143 | Dyrk1A Phosphorylates α-Synuclein and Enhances Intracellular Inclusion Formation | 1.3 | 112 | Citations (PDF) |
| 144 | Unique Tauopathy in Fukuyama-Type Congenital Muscular Dystrophy | 1.4 | 9 | Citations (PDF) |
| 145 | Tau-Positive Fine Granules in the Cerebral White Matter: A Novel Finding Among the Tauopathies Exclusive to Parkinsonism-Dementia Complex of Guam | 1.4 | 21 | Citations (PDF) |
| 146 | Four-repeat tau-positive Pick body-like inclusions are distinct from classic Pick bodies | 5.8 | 7 | Citations (PDF) |
| 147 | Phosphorylation of FTDP-17 Mutant tau by Cyclin-dependent Kinase 5 Complexed with p35, p25, or p39 | 1.3 | 38 | Citations (PDF) |
| 148 | Inhibition of Heparin-induced Tau Filament Formation by Phenothiazines, Polyphenols, and Porphyrins | 1.3 | 519 | Citations (PDF) |
| 149 | Ubiquitination of α-Synuclein† | 1.5 | 135 | Citations (PDF) |
| 150 | Accumulation of phosphorylated a-synuclein in the brain and peripheral ganglia of patients with multiple system atrophy | 5.8 | 117 | Citations (PDF) |
| 151 | Identification of amino-terminally cleaved tau fragments that distinguish progressive supranuclear palsy from corticobasal degeneration | 4.6 | 183 | Citations (PDF) |
| 152 | Alterations in human tau transcripts correlate with those of neurofilament in sporadic tauopathies | 1.3 | 39 | Citations (PDF) |
| 153 | Phosphorylation of α-synuclein characteristic of synucleinopathy lesions is recapitulated in α-synuclein transgenic Drosophila | 1.3 | 132 | Citations (PDF) |
| 154 | Neuronal and glial inclusions in frontotemporal dementia with or without motor neuron disease are immunopositive for p62 | 1.3 | 83 | Citations (PDF) |
| 155 | α-Synuclein Accumulates in Purkinje Cells in Lewy Body Disease but not in Multiple System Atrophy | 1.4 | 76 | Citations (PDF) |
| 156 | Accumulation of Phosphorylated α-Synuclein in Aging Human Brain | 1.4 | 337 | Citations (PDF) |
| 157 | Biochemical Characterization of the Core Structure of α-Synuclein Filaments | 1.3 | 246 | Citations (PDF) |
| 158 | Phosphorylated α-Synuclein Is Ubiquitinated in α-Synucleinopathy Lesions | 1.3 | 404 | Citations (PDF) |
| 159 | A clinical and neuropathological study of an unusual case of sporadic tauopathy. A variant of corticobasal degeneration? | 1.3 | 17 | Citations (PDF) |
| 160 | α-Synuclein is phosphorylated in synucleinopathy lesions | 12.6 | 2,095 | Citations (PDF) |
| 161 | Misfolded proteinase K–resistant hyperphosphorylated α-synuclein in aged transgenic mice with locomotor deterioration and in human α-synucleinopathies | 6.6 | 201 | Citations (PDF) |
| 162 | Misfolded proteinase K–resistant hyperphosphorylated α-synuclein in aged transgenic mice with locomotor deterioration and in human α-synucleinopathies | 6.6 | 305 | Citations (PDF) |
| 163 | Tau accumulation in a patient with pallidonigroluysian atrophy | 1.3 | 14 | Citations (PDF) |
| 164 | Co-localization of α-synuclein and phosphorylated tau in neuronal and glial cytoplasmic inclusions in a patient with multiple system atrophy of long duration | 5.8 | 81 | Citations (PDF) |
| 165 | Age-Related Amyloid β Deposition in Transgenic Mice Overexpressing Both Alzheimer Mutant Presenilin 1 and Amyloid β Precursor Protein Swedish Mutant Is Not Associated with Global Neuronal Loss | 2.8 | 232 | Citations (PDF) |
| 166 | Structure of tau exon 10 splicing regulatory element RNA and destabilization by mutations of frontotemporal dementia and parkinsonism linked to chromosome 17 | 5.2 | 254 | Citations (PDF) |
| 167 | The Tauopathies | 2.8 | 55 | Citations (PDF) |
| 168 | Frontotemporal Dementia and Corticobasal Degeneration in a Family with a P301S Mutation in Tau | 1.4 | 424 | Citations (PDF) |
| 169 | Tau proteins with FTDP‐17 mutations have a reduced ability to promote microtubule assembly | 1.8 | 469 | Citations (PDF) |
| 170 | α-Synuclein in filamentous inclusions of Lewy bodies from Parkinson’s disease and dementia with Lewy bodies | 5.2 | 3,108 | Citations (PDF) |
| 171 | Alzheimer-like Changes in Microtubule-associated Protein Tau Induced by Sulfated Glycosaminoglycans | 1.3 | 198 | Citations (PDF) |
| 172 | Somatodendritic localization of phosphorylated tau in neonatal and adult rat cerebral cortex | 1.2 | 92 | Citations (PDF) |
| 173 | Dephosphorylation of fetal-Tau and Paired Helical Filaments-Tau by Protein Phosphatases 1 and 2A and Calicineurin1 | 1.4 | 44 | Citations (PDF) |
| 174 | Dephosphorylation of fetal tau protein and its implication for Alzheimer’s disease. | 1.4 | 0 | Citations (PDF) |
| 175 | Proline-directed and Non-proline-directed Phosphorylation of PHF-tau | 1.3 | 547 | Citations (PDF) |
| 176 | Hyperphosphorylation of Tau in PHF | 2.4 | 215 | Citations (PDF) |
| 177 | Ubiquitin is conjugated with amino-terminally processed tau in paired helical filaments | 8.2 | 336 | Citations (PDF) |
| 178 | Immunochemical evidence that fragments of phosphorylated MAP5 (MAP1B) are bound to neurofibrillary tangles in Alzheimer's disease | 8.2 | 111 | Citations (PDF) |
| 179 | Identification of β protein precursor in newborn rat brain | 1.5 | 32 | Citations (PDF) |
| 180 | Effect of L-DOPA/Benserazide on Propagation of Pathological α-Synuclein | 2.0 | 21 | Citations (PDF) |
| 181 | α-synuclein strains that cause distinct pathologies differentially inhibit proteasome | 1.0 | 67 | Citations (PDF) |
| 182 | Title is missing! 0 | | 1 | Citations (PDF) |
| 183 | Animal models of tau propagation in Alzheimer's disease | 1.9 | 5 | Citations (PDF) |
| 184 | Argyrophilic grain disease: epidemiology and association with cognitive decline and parkinsonism | 2.0 | 7 | Citations (PDF) |
| 185 | Hindering tau fibrillization by disrupting transient precursor clusters | 1.9 | 1 | Citations (PDF) |
| 186 | Co-aggregation of annexin A11 and TDP-43 in FTLD/MND with primary lateral sclerosis phenotype | 3.3 | 4 | Citations (PDF) |
| 187 | Repetitive mild traumatic brain injury with the closed-head impact model of engineered rotational acceleration (CHIMERA) promotes tau pathology in tau transgenic mice and its propagation in brains injected with tau fibrils | 3.3 | 1 | Citations (PDF) |
| 188 | Impaired $$\alpha$$-Synuclein aggregate clearance in neuronal cells drive their spread to microglia through tunneling nanotubes | 10.8 | 7 | Citations (PDF) |
| 189 | α-Synuclein aggregates induce mitochondrial damage and trigger innate immunity to drive neuron–microglia communication | 10.8 | 2 | Citations (PDF) |
| 190 | Enhancing heme synthesis attenuates pathological α-synuclein propagation in vivo | 3.3 | 0 | Citations (PDF) |
| 191 | Mitochondrial alterations in pellagra-associated central chromatolysis: Comparison with ballooned achromatic neurons of other etiologies | 1.4 | 0 | Citations (PDF) |
